dc.contributor.author |
Ozyurt, H |
|
dc.contributor.author |
Irmak, MK |
|
dc.contributor.author |
Akyol, O |
|
dc.contributor.author |
Sogut, S |
|
dc.date.accessioned |
2022-03-07T11:46:07Z |
|
dc.date.available |
2022-03-07T11:46:07Z |
|
dc.date.issued |
2001 |
|
dc.identifier.uri |
http://hdl.handle.net/11616/54502 |
|
dc.description.abstract |
Oxygen-derived free radicals have been implicated in the pathogenesis of renal injury after ischaemia-reperfusion. Caffeic acid phencthyl ester (CAPE), an active component of propolis extract, exhibits antioxidant properties. To investigate whether treatment with either CAPE or alpha-tocopherol modifies the levels of the endogenous indices of oxidant stress, we examined their effects on an in vivo model of renal ischaemia-reperfusion injury in rats. CA-PE at 10 mu mol kg(-1) or alpha-tocopherol at 10 mg kg(-1) was administered intraperitoneally before reperfusion. Acute administration of both CAPE and alpha-tocopherol altered the indices of oxidative stress differently in renal ischaemia-reperfusion injury. Copyright (C) 2001 John Wiley & Sons, Ltd. |
|
dc.source |
CELL BIOCHEMISTRY AND FUNCTION |
|
dc.title |
Caffeic acid phenethyl ester changes the indices of oxidative stress in |
|
dc.title |
serum of rats with renal ischaemia-reperfusion injury |
|